The body's initial immune response to trauma is inflammation, which is triggered by the release of communication proteins in the affected area. Every time we eat food with chemicals, emulsifiers, GMOs, or anything else that is not a natural food or herb product, the body will cause inflammation, which will cause damage to your cells. These damaged cells can then break away, becoming rogue cells. These rogue cells can then metastasize, becoming plaque in the bloodstream. This will lead to premature aging, aches and pains, and even chronic illness.
Uncontrolled inflammation will lead to the formation of rogue cells that can cause severe damage to the body. These cells produce interleukins, which prevent cells from destroying themselves when damaged cells cannot repair their internal damage. These rogue cells can evade the immune system's response, leading to angiogenesis, where pro-inflammatory communication proteins cause blood vessel growth to sustain an expanding oxygen need. As these cells divide, pro-inflammatory interleukins signal them to break loose and migrate through the blood, causing metastasis. These metastasized cells are not recognized as "bad" by the immune system, making it difficult to destroy and eliminate them. Around thirty different communication proteins are created, providing a multilayered approach to healing. C reactive proteins, which grow new capillaries for better blood flow and divide cells, are used to repair damaged cells. NF-kB triggers these pro-inflammatory conditions, which are crucial for the beneficial repair of the inflamed area.
Over the past eighty years, numerous animal and human trials have explored the impact of eating less on our lifespan. The first test found that eating 35% less than normal increased the lifespan by 46%! This is because humans have faced periods of food scarcity throughout history, so our bodies are made for it. If we never fast, we never clean out the body's bad cells, so they will just continue to pile up, making you appear older and more bloated than you should. Our DNA is our library of blueprints to make proteins. There are actually specific proteins released during starvation that slow our growth and development; this is nature's way of allowing us to wait for a better time when food is plentiful. When we eat more than we need, our DNA reverses this process and produces other "controlling" proteins that cause us to "age" more rapidly. Then throw in all the other toxic seed oils, emulsifiers, preservatives, and chemicals in our food, and you have an inflammation epidemic! This is why 60% of people over 60 have chronic illnesses. We are simply eating too much toxic food.
Did you know that your body's response to the chemicals you ingest can impact your health more than you realize? It turns out that fat cells play a crucial role in this process, emitting a protein complex called NF-kB that influences DNA transcription. This revelation has significant implications for your overall well-being.
The Inflammatory Culprit: NF-kB and Fat Cells:Fat cells, also known as adipocytes, do more than just store energy. They release a protein complex called NF-kB, which wields power over DNA transcription. In simpler terms, NF-kB controls how genes are expressed in response to different stimuli, including the chemicals you consume.
The Domino Effect: Chemical Ingestion and Inflammation:Your body is remarkably adaptive, constantly responding to the barrage of chemicals it encounters. When a foreign substance enters your bloodstream, your immune system kicks into gear with a pro-inflammatory response. This inflammation helps identify and eliminate these intruders. However, chronic exposure to these chemicals can throw this balance off-kilter.
The Ongoing Battle: Inflammation in Fat Tissue:Imagine sipping alcohol, taking pharmaceutical drugs, or even inhaling cigarette smoke. Thousands of toxic chemicals flood your bloodstream, triggering a pro-inflammatory response. Fat tissue becomes a battleground, with immune cells mounting a defense against these invaders. Unfortunately, constant exposure without respite leads to prolonged inflammation, causing wear and tear on your body.
The Toll on Your Health:While our bodies are designed to handle occasional inflammatory challenges, continuous exposure to toxins disrupts the delicate equilibrium. Your body needs a breather to effectively remove these toxins and reduce inflammation. Otherwise, this unrelenting assault can lead to organ dysfunction and premature aging.
Give Your Body a Break:Your health is at stake, and the solution lies in moderation. Providing your body with periods of toxin-free respite is essential. This allows the inflammatory response to subside naturally, preventing long-term damage. Remember, your body is a remarkable system, but even the strongest system needs time to recuperate.
The Impact of Excessive Blood Sugar on Heart Health:Modern research has shed light on the detrimental effects of consistently high blood sugar levels on heart health. Excess sugar acts like sticky glue within the bloodstream, thickening the blood and attracting immune cells called macrophages. This process triggers inflammation and can lead to swelling of heart tissues. As arteries narrow due to plaque buildup, blood flow is compromised, depriving organs and tissues of oxygen. Moreover, the risk of arterial rupture and leakage increases, potentially causing strokes and heart attacks.
Prevention: Your Pathway to Heart Health:While the adverse effects of elevated blood sugar levels may seem daunting, the good news is that prevention is within reach. By taking charge of your diet and blood sugar levels, you can mitigate the risk of heart-related issues and enjoy a healthier life. Here are some actionable steps to consider:
Ever wondered what keeps our bodies running smoothly? Meet mitochondria, the microscopic engines inside our cells. They play a big role in how we age and affect our overall health. Let's explore how these little powerhouses impact our bodies in simple terms.
Mitochondria: Our Cellular Power Plants:Think of mitochondria as mini power plants. They take the food we eat and turn it into energy that fuels everything we do. This energy molecule, called ATP, is like the body's battery.
The Trouble with Escaping Electrons:Sometimes, in their energy-making process, mitochondria let a few electrons escape. This causes a bit of trouble, as these "escaped" electrons can damage our cells and lead to aging.
Protectors and Helpers:To fight this trouble, our bodies have defenders called antioxidants. They neutralize the harmful effects of those escaped electrons. There are also "helpers" called chaperone proteins that bring these defenders to where they're needed.
Mitophagy: Recycling Old Engines:Our body is smart—it knows when some mitochondria get old or damaged. It recycles them through a process called mitophagy. But as we age, this recycling process isn't as efficient, which can cause problems.
Effects on Our Body:When mitochondria struggle to make energy, our muscles weaken, our hearts and lungs lose power, and even our thinking can slow down. Imagine these engines losing steam over time.
Kidney Quandary:Our kidneys also feel the impact. High sugar levels can mess with their filtering job, leading to chemical imbalances and more aging symptoms.
Conclusion:Mitochondria are like the little workers that keep our body machinery running. Taking care of them by eating well, staying active, and managing stress can help us age gracefully. While we can't stop time, we can give our cells' powerhouses the support they need for a healthier, more vibrant life.
Sugar and fatty acids in our blood cross-link with proteins in our blood or vessels, creating sticky, gooey junk molecules that clog up our body's structure and functions. These molecules are essential for powering our mitochondria, which produce ATP that powers all cellular biological processes. However, too much sugar and fat become oxidized, leading to oxidative stress and the accumulation of sticky junk molecules. This spiral continues to build, creating bigger masses of sticky junk molecules that clog our structures and functions, leading to old age.These junk molecules can form inside and outside our cells, leading to aging symptoms and disease states such as diabetic kidney, nerve, and retinal damage. Plaques form within arteries, and junk glycation forms around nerve cells, leading to dizziness, confusion, and loss of memory. This causes cells to die prematurely, leading to a lack of neurotransmitters and symptoms of trembling and stiffening of body muscles.As these glycated proteins link across the tissue, the tissue becomes bound up and no longer flexible, causing symptoms like the elderly's movement compared to young adults' flexibility. Removing junk glycated proteins from our system could alleviate old age symptoms and rejuvenate our structure and functions. This would be like vacuuming the rug regularly and wiping the cobwebs off the ceiling every day.Our cells are based on our current metabolism, and research chemists have found that if we eat a lot of food daily, our life is shorter than that of a person who eats less. However, both overeaters and undereaters eat roughly the same amount of food in their respective lifetimes.In conclusion, removing junk glycated proteins from our system can alleviate old age symptoms and rejuvenate our structure and functions.
Our immune systems play a crucial role in combating various infections and diseases. The immune system only allows a certain number of T and B cells to be dedicated to specific rogue proteins. After destroying the invading proteins, the T and B cells scale back, leaving some with a memory for future attacks. The rest are re-cycled and reprogrammed for new threats. Some viruses cannot be completely eliminated, and a small amount hides in the DNA to be expressed at a later time. T and B cells mount successive attacks over and over again, but never completely defeating the virus. As a result, they become resolute to stay, fight, and resist being recycled. This leads to an ever-growing steady-state force, depleting the immune system's limited resources.
These anergic cells become crippled and weakened, losing other internal capabilities. When T cells divide to quickly respond to environmental threats, they can be diverted from alerts and stop copying. This is due to the protection mechanism developed by the cell to differentiate between an accidental break in the DNA and the actual end of it. The reproducing mechanism breaks off a little bit of the telomere end, causing the DNA to no longer be replicated (the Hayflick limit).
Our immune cells constantly divide to produce more cells for different fights against invading bacteria and viruses. Within the DNA, a gene encodes for a special catalyst called Telomerase, which can lengthen the telomeres of the DNA. Unfortunately, anergic T and B cells become dysfunctional and lose their ability to lengthen their telomeres. When cells cannot continue to divide due to short telomeres, they are termed senescence, or old age. This results in the immune system becoming weaker over time, leading to memory loss and the formation of amyloid plaques that cut off neuron cells.
Senescent cells are old cells that cannot divide anymore and are discovered by Professor Hayflick. These cells, which grow well for a period but suddenly stop reproducing, change shape, and take on colors that younger cells don't have. Some cells, like skin, joints, and connective tissue, can reproduce by cell division, while others, like heart and brain cells, do not divide again. The Hawflick limit is the number of times cells split before they stop dividing, which is set by the telomeres on the end of the DNA getting shorter every time the cell divides. This is a protective mechanism to prevent DNA from being knitted together by mistake and to prevent a large improper mass of cells from growing to a large improper mass of cells, which would cause detriment to the body.
The Hawflick limit is a protective mechanism to prevent DNA from being knitted together by mistake and to prevent cancer from growing to a large improper mass. However, senescent cells occur randomly, interspersed throughout normal tissues and organs in the body. These cells are more active than previously thought, producing many complex proteins that stimulate themselves to a younger state. These chemicals can promote the activity of surrounding neighboring cells, which could potentially lead to cancer.
One of the most abundant proteins is epidermal growth factor, which causes cell division in adjacent cells. Another overproduced protein is matrix metalloproteinase (MMP), which is used to tear down old cellular structures, allowing new growth but releasing rouge cells that were embedded in the destroyed structure. This process allows these cells to migrate to other areas of the body to grow.
Lastly, old senescent cells release an overdose of two proteins that promote the growth of new blood vessels, called angiogenesis. Rouge cells that are growing rapidly need fresh blood, which new capillaries provide.
